In Vivo Modelling of ATP1A3 G316S-Induced Ataxia in C. elegans Using CRISPR/Cas9-Mediated Homologous Recombination Reveals Dominant Loss of Function Defects.

author

  • Hart, Anne Professor of Neuroscience, NIH Graduate Partnership Program (NIH GPP) Director, Graduate Program Director for the Neuroscience Graduate Program